basal ganglia calcification oor Spaans
basal ganglia calcification
Vertalings in die woordeboek Engels - Spaans
Síndrome de Fahr
en
basal ganglia disease characterized by the presence of abnormal calcium deposits of unknown cause in the brain; has symptom dementia, psychosis, mood swings and loss of acquired motor skills
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The brain computed tomography showed basal ganglia calcifications.
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Clinical Case Report: A case of a 57-year-old male patient, with right ipsilateral hemiparesia, basal ganglia calcifications and spinal meningeal cysts.
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Ipsilateral hemiparesia and calcifications in the basal ganglia: Case Report
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Calcifications in the basal ganglia can be physiological or pathological findings in up to 0.6% of patients studied with neuroimagery.
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The imaging abnormalities found comprised cortical atrophy (cortical or central), basal ganglia calcification, and demyelinizing disorders.
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Discussion: Patients with basal ganglia calcifications have extrapyramidal symptoms (20-30%) such as parkinsonism or coreoatetosis, seizures, and neuropsychiatric disorders.
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In 1984, Jean Aicardi and Francoise Goutières described eight children from five families presenting with a severe early onset encephalopathy, which was characterized by calcification of the basal ganglia, abnormalities of the cerebral white matter and diffuse brain atrophy.
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Calcification is detected on x-rays of the subcutaneous or paraspinal basal ganglia of the brain.
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To report a case of 46-year-old male patient with structural focal epilepsy and mild cognitive impairment who presented to the emergency department with 15 days history of neurological deterioration. Simple brain CT revealed multiple brain calcifications in basal ganglia, white matter and cerebellum, along with hypocalcemia, hyperphosphatemia, hypomagnesemia and normal PTH. A diagnosis of Fahr syndrome associated with pseudohypoparathyroidism was considered.
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In both cases it was found an increase of pterines in the cerebrospinal fluid, mainly neopterine, with calcifications in the basal ganglia.
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She presented right sided hypotonia, right upper limb winding movements, hypocalcemia, hyperphosphatemia and low parathormone levels; tomography and magnetic resonance showed bilateral calcifications in brain basal ganglia and cerebellum.
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Case report: Female 48 year-old patient who had thyroidectomy 27 years before, received levothyroxine, and who was attended for chorea, ballistic involuntary movements and progressive difficulty in walking. She presented right sided hypotonia, right upper limb winding movements, hypocalcemia, hyperphosphatemia and low parathormone levels; tomography and magnetic resonance showed bilateral calcifications in brain basal ganglia and cerebellum. She was treated with calcium citrate, calcitriol and risperidone with complete recovery.
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Congenital infections can affect multiple organ systems, and many are associated with specific brain damage, including microcephaly, calcifications (predominantly periventricular, but also in the basal ganglia and in cerebral parenchyma), ventriculomegaly, neuronal migration disorders (pachygyria, polymicrogyria, lissencephaly, and schizenchephaly), cerebellar hypoplasia, and white matter anomalies (8).
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Congenital infections can affect multiple organ systems, and many are associated with specific brain damage, including microcephaly, calcifications (predominantly periventricular, but also in the basal ganglia and in cerebral parenchyma), ventriculomegaly, neuronal migration disorders (pachygyria, evaluation of new cases are important to describe the phenotypic spectrum of potential Zika virus-associated congenital infections.
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Computed tomography scans and transfontanellar cranial ultrasounds showed a consistent pattern of widespread brain calcifications, mainly in the periventricular, parenchymal, and thalamic areas, and in the basal ganglia, and was associated in approximately one third of cases with evidence of cell migration abnormalities (e.g., lissencephaly, pachygyria).
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