medullary carcinoma oor Spaans
medullary carcinoma
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carcinoma medular
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Chief among these is medullary carcinoma.
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Approximately 75% of medullary carcinomas occur sporadically, but 25% are familial in origin.
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Medullary carcinoma of the thyroid can have a variety of histologic and cytologic forms.
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Medullary carcinoma has a natural history that is more aggressive than papillary or follicular thyroid carcinoma.
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In addition, medullary carcinoma can spread via the blood to the lungs and viscera.
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Surgery is the only effective therapy for medullary carcinoma.
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Total thyroidectomy and a central neck dissection are recommended for all patients with medullary carcinoma.
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Once medullary carcinoma is suspected, calcitonin immunohistochemistry can confirm the FNA diagnosis.
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Medullary carcinoma is a morphologically similar neoplasm that is seen in patients with sickle cell trait.
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Medullary carcinoma is derived from the parafollicular cells.
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[Reproduced with permission from Wells S, Franz C: Medullary carcinoma of the thyroid.
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medullary carcinoma is also associated with sickle cell trait and occurs predominantly in young adults and children.
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There is no role of 131I or chemotherapy in the treatment of medullary carcinoma.
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Eighty percent of medullary carcinomas are sporadic, and the remainder are familial.
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In medullary carcinomas of the thyroid, the presence of amyloid is an essential diagnostic feature.
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Mutation of the RET proto-oncogene is seen in most cases of familial medullary carcinoma.
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Ten year survival in differentiated carcinomas was 92%, 70% five year survival in medullary carcinoma, with no survival in anaplasic carcinomas.
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The strongest correlation between tumor cell nuclear DNA content and survival was obtained in the group with papillary carcinoma, followed by follicular and medullary carcinoma.
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Recently, however,131I uptake has been reported in metastases of “medullary carcinoma” and, consequently, some authors have suggested that these tumors can be treated with radioactive iodine.
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Somatostatin (5 × 10−6M) decreased basal and TSH stimulated AC activity below basal levels in both normal and neoplastic thyroid tissue (including papillary, follicular, and medullary carcinomas).
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Apart from 4 patients with small nodules of medullary carcinoma, the values in all patients with differentiated or anaplastic carcinomas were classified as group III, IV, or IV+.
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Neuroendocrine carcinomas may metastasize to the thyroid and be confused with medullary thyroid carcinoma.
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In our 5 patients who were treated by total thyroidectomy, the diagnosis of medullary carcinoma was made by experienced pathologists using immunohistochemical staining methods (calcitonin and/or thyroglobulin), peroxidase-antiperoxidase techniques, and electron microscopy.
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We have studied a series of 22 human medullary carcinomas (MCTs), both primary and metastatic, using immunocytochemistry (ICC) to localize calcitonin and somatostatin peptide and in situ hybridization (ISH) to localize calcitonin and somatostatin mRNA.
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The management of medullary thyroid carcinoma is surgical, as these tumors do not take up radioiodine.
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