stellate cell of liver oor Spaans
stellate cell of liver
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célula de Kupffer
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Scientists have investigated the role of hepatic stellate cells on a common symptom of excess iron, fibrosis of the liver.
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Liver fibrosis and cirrhosis is caused by an interplay of hepatocyte toxicity and hepatic stellate cells activation which caused a deposition of collagen, along with changes in the liver sinusoidal endothelial cells which lose their specific fenestrated membrane.
La Comisión tendrá en cuenta especialmente cualquier evaluación de impacto de la medida propuesta que el Estado miembro pueda haber realizadocordis cordis
This bioreactor supports the interaction of hepatocytes with the non-parenchymal fraction of the liver such as hepatic stellate cells (HSCs) and liver sinusoidal endothelial cells (LSECs).
Son de aplicación las siguientes disposiciones de la Ley relativa al comercio y los pagos exteriores (Aussenwirtschaftsgesetz, en lo sucesivo la AWG) y del Decreto relativo al comercio y los pagos exteriores (Außenwirtschaftsverordnung, en lo sucesivo el AWV), adoptados el # de diciembre decordis cordis
A third achievement, was, by using cocultures of hepatocyte and stellate cells, the creation of a unique in vitro model to study repeated dose toxicity ultimately leading to liver fibrosis was developed.
Revisamos sus signos vitales todo el caminocordis cordis
After a liver injury activation of hepatic stellate cells (HSC) and their conversion to contractile, proliferative and fibrogenic cells responsible for the production of major extracellular matrix proteins (MEC) are the current focuses of investigation. However, the prospect of reversing hepatic fibrosis through the discovery of new antifibrotic therapies is even more attractive to researchers.
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The major cellular event in the development and progression of liver cirrhosis is the activation of hepatic stellate cells.
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In addition, one of the consequences of untreated viral hepatitis is liver fibrosis, which is the formation of scar tissue in reaction to liver injury by the activation of hepatic stellate cells (HSCs).
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They occur in GRANULATION TISSUE and also in pathological processes such as FIBROSIS.Epithelium: One or more layers of EPITHELIAL CELLS, supported by the basal lamina, which covers the inner or outer surfaces of the body.Hepatic Stellate Cells: Perisinusoidal cells of the liver, located in the space of Disse between HEPATOCYTES and sinusoidal endothelial cells.Connective Tissue Growth Factor: A CCN protein family member that regulates a variety of extracellular functions including CELL ADHESION; CELL MIGRATION; and EXTRACELLULAR MATRIX synthesis.
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In 1985, GRX, a continuous cell line representative of hepatic stellate cells, was established from fibro-granulomatous lesions induced in mouse liver by schistosomal infection2, and its biological and biochemical characteristics were determined3,4,5.
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Liver cell death and inflammatory responses lead to the activation of stellate cells which play a pivotal role in hepatic fibrosis.
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The role of the protein scavenger Spa in the interaction between macrophages, hepatic stellate cells and hepatocytes in liver cancer (Dr Maria Rosa Sarrías Fornés)
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According to research at the University of Bonn, Germany, the endocannabinoid N-arachidonoyl dopamine (NADA) dose-dependently induced cell death in human hepatic stellate cells, but not in normal liver cells.
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The process of switching one differentiated cell type into another cell type with a different form and function, such as transdifferentiation from hepatic stellate cells to myofibroblastic cells in liver fibrogenesis.
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Hepatic fibrosis can be regarded as an integrated and highly dynamic cellular response to chronic liver damage.10 Its progression is characterized by the perpetuation of necrosis of the parenchyma, chronic hepatitis, and qualitative and quantitative alterations in the composition of the extracellular matrix (ECM), while the activation of hepatic stellate cells (HSC) and the participation of macrophages and Kupffer cells predominate at the cellular level.10–12 At the molecular level, growth factors, cytokines, chemokines, changes in ECM organization and composition, as well as oxidative stress-related molecules, are seen to play an important pathologic role.10–12 The participation of oxidative stress in almost all clinical and experimental conditions of chronic liver disease of different etiologies, including alcohol consumption, has been demonstrated.13–15
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