hypercalcemia oor Indonesies

hypercalcemia

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en
(medicine) The presence of an unusually high concentration of calcium in the blood

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Hiperkalsemia

en
Human disease
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Prominent U waves are most often seen in hypokalemia but may be present in hypercalcemia, thyrotoxicosis, or exposure to digitalis, epinephrine and Class 1A and 3 antiarrhythmics, as well as in congenital long QT syndrome, and in the setting of intracranial hemorrhage.
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Consumption of calcium chloride can lead to hypercalcemia.
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Calcitonin can be used therapeutically for the treatment of hypercalcemia or osteoporosis.
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Increased magnesium lowers calcium and can either prevent hypercalcemia or cause hypocalcemia depending on the initial level.
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In Zollinger–Ellison syndrome and hypercalcemia, there are increased gastrin levels, leading to excess gastric acid production, which can cause gastric ulcers.
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It can be usually prevented by potassium supplements or by combining hydrochlorothiazide with a potassium-sparing diuretic Other disturbances in the levels of serum electrolytes including hypomagnesemia (low magnesium), hyponatremia (low sodium), and hypercalcemia (high calcium) Hyperuricemia, high levels of uric acid in the blood Hyperglycemia, high blood sugar Hyperlipidemia, high cholesterol and triglycerides Headache Nausea/vomiting Photosensitivity Weight gain Gout Pancreatitis These side effects increase with the dose of the medication and are most common at doses of greater than 25 mg per day.
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It is also used for liver cirrhosis, kidney impairment, nephrotic syndrome, in adjunct therapy for swelling of the brain or lungs where rapid diuresis is required (IV injection), and in the management of severe hypercalcemia in combination with adequate rehydration.
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Aromatic amino acids are particularly powerful stimuli for gastrin release. hypercalcemia (via calcium-sensing receptors) Gastrin release is inhibited by: the presence of acid (primarily the secreted HCl) in the stomach (a case of negative feedback) somatostatin also inhibits the release of gastrin, along with secretin, GIP (gastroinhibitory peptide), VIP (vasoactive intestinal peptide), glucagon and calcitonin.
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Acquired nephrogenic diabetes insipidus in rats can be caused impaired regulation of aquaporin-2 due to administration of lithium salts, low potassium concentrations in the blood (hypokalemia) and high calcium concentrations in the blood (hypercalcemia).
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