Infection with the herpes simplex virus, commonly known as herpes, can be due to either herpes simplex virus type 1 (HSV-1) or herpes simplex virus type 2 (HSV-2).
Two clinical trials are currently assessing the efficacy of preventing HIV through the suppression of herpes simplex virus type # which is known to increase the risk of HIV transmission and acquisition
Two clinical trials are currently assessing the efficacy of preventing HIV through the suppression of herpes simplex virus type 2, which is known to increase the risk of HIV transmission and acquisition.
People who already have HSV-1 infection are not at risk of getting it again, but they are still at risk of acquiring herpes simplex virus type 2 (HSV-2) genital infection (see below).
Data on men aged 15 to 64 years were derived from the 2007 Kenya AIDS Indicator Survey, which involved interviews and blood collection to test for HIV and herpes simplex virus 2 (HSV-2).
Studies of early-generation microbicides have similarly failed to detect a prevention benefit, and disappointing results were reported on the HIV prevention potential of female diaphragms and community-based acyclovir treatment for herpes simplex virus type 2.
The thinning of vaginal mucosa with age may play a role; for both sexes, the prevalence of antibodies against herpes simplex virus 2 increases with age,11 indicating continual risky sexual behaviour and enhanced risk of HIV transmission.
28 October 2015 ¦ GENEVA - More than 3.7 billion people under the age of 50 – or 67% of the population – are infected with herpes simplex virus type 1 (HSV-1), according to WHO’s first global estimates of HSV-1 infection published today in the journal PLOS ONE.
Anogenital herpes is caused mainly by herpes simplex type # virus ( # ) # prevalence varies widely, with generally higher rates in developing than in developed countries and in urban than in rural areas
A World Health Organization (WHO) report explains that one category of ultraviolet light, known as UVB, “appears to reduce the effectiveness of the immune system —in the case of cold sores it can no longer keep the virus Herpes simplex under control which results in re-activation of the infection.”