Recent information indicates that endosulfan mimics non-uterotrophic E actions, strengthening the hypothesis that endosulfan is a widespread xenoestrogen (Varayoud et al # ), acts via a membrane version of the estrogen receptor-α on pituitary cells and can provoke Ca++ influx via L-type channels, leading to prolactin (PRL) secretion (Watson et al # ), and is also anti-progestative (Chatterjee et al
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