Organomegaly oor Spaans

Organomegaly

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Organomegalia

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organomegaly

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The abnormal enlargement of an organ

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The remainder of the physical examination should search for signs of ascites, pleural effusion, and organomegaly.
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Physical examination showed normal vital signs, but his bowel sounds were more intense and frequent (40/min) than normal, and he suffered mesogastric pain on palpation. There were no masses or organomegaly.
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No organomegaly was noted, and bowel sounds were normal.
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Simpson-Golabi-Behmel syndrome (SGBS) is a rare overgrowth syndrome clinically characterized by multiple congenital abnormalities, pre/postnatal overgrowth, distinctive craniofacial features, macrocephaly, and organomegaly.
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Organomegaly, especially thyromegaly, is common, but hyperthroidism is exceedingly rare unless the tumor cosecretes TSH.
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Organomegaly is the abnormal enlargement of organs.
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Due to the missing or defective iduronate-#-sulfatase enzyme in patients with Hunter syndrome, glycosaminoglycans progressively accumulate in the cells, leading to cellular engorgement, organomegaly, tissue destruction, and organ system dysfunction
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No palpable organomegaly.
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Simpson-Golabi-Behmel syndrome (SGBS) is a rare overgrowth syndrome clinically characterized by multiple congenital abnormalities, pre/postnatal overgrowth, distinctive craniofacial features, macrocephaly, and organomegaly.
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Organomegaly is seldom prominent early in the course.
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It has systemic compromise, mainly neurologic disease and organomegaly.
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No organomegaly was evident on abdominal exam, and the genitalia was within normal limits.
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POEMS (polyneuropathy, organomegaly, endocrinopathy, monoclonal gammopathy, and skin changes) syndrome is a rare paraneoplastic condition associated with a plasma cell dyscrasia of early or late stage.
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No skin lesions, organomegaly or other clinical alterations.
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POEMS, polyneuropathy, organomegaly, endocrinopathy, M protein, skin changes.
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Laboratory tests unveiled a monoclonal gammopathy. The diagnosis was an “incomplete” POEMS syndrome (polyneuropathy, organomegaly, endocrinopathy, M component and skin changes).
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Abdominal examination should note areas of tenderness, presence of peritoneal findings (eg, guarding, rigidity, rebound), and any masses or organomegaly.
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For patients with loss of imprinting at ICR2, an abdominal ultrasound examination is performed at the time of clinical or molecular diagnosis; only patients with organomegaly or severe hemihyperplasia require surveillance by ultrasound scans. Monthly clinical examinations are performed for the first 2 years, followed by clinical examinations every 3 to 6 months until age 6 years.
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Organomegaly: increased size of internal organs, affecting the spleen and liver.
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The acronym, which was coined by Bardwick in 1980, refers to several, but not all, of the features of the syndrome: polyradiculoneuropathy, organomegaly, endocrinopathy, monoclonal PCD, and skin changes.
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Abdominal mass or organomegaly
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For patients with loss of imprinting at ICR2 (ICR2-LoM), an abdominal ultrasound examination is performed at the time of clinical or molecular diagnosis; only patients with organomegaly or severe hemihyperplasia require surveillance by ultrasound scans.
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Syndrome, POLYNEUROPATHIES), organomegaly, endocrinopathy, monoclonal gammopathy, and pigmentary skin changes.
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Clinics: organomegaly; frequent CNS involvement (5%); high WBC (above 50 x 109/l in 40%).
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The entire body (particularly over the spine, bones, joints, abdomen, and thyroid) is palpated for areas of tenderness, swelling, or organomegaly; digital rectal examination and pelvic examination are included.
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